The diosmin antioxidant effect: an integrative review

Main Article Content

Amanda Francielle Santos
Rafaela Ribeiro Machado
José Marden Mendes Neto
Míriam Geisa Virgens Menezes
Gilmara Beatriz Andrade da Silva
Sandra Lauton Santos

Abstract

Oxidative stress is related to the development of multiple diseases and some studies objective to identify the effect of natural substances in lesions caused by it. One of these substances is Diosmin, a flavonoid found in citric fruits. The objective of this study was to identify through the literature the anti-oxidative effect of Diosmin. It is a descriptive and bibliographic study of integrative revision, based on articles which discussed the researched topic published between 2013 and 2017 using the electronic database PubMed and Bireme. 9 articles were analyzed. The studies evaluated show that the use of Diosmin rises the activity of enzymes which fight against oxidative stress, as catalase, superoxide dismutase and glutathione. There was reduction of the concentration of malondialdehyde. It was proved with this study that Diosmin contributes to the fight against lesions caused by the unbalance between the redox status, stimulating the rise of activity of enzymatic anti-oxidants and reducing the production of malondialdehyde, collaborating to the reduction of damage to tissues generated by stress oxidative.

Downloads

Download data is not yet available.

Article Details

How to Cite
Santos, A. F., Machado, R. R., Mendes Neto, J. M., Menezes, M. G. V., Silva, G. B. A. da, & Santos, S. L. (2018). The diosmin antioxidant effect: an integrative review. ABCS Health Sciences, 43(3). https://doi.org/10.7322/abcshs.v43i3.1024
Section
Review Articles
Author Biographies

Amanda Francielle Santos, Curso de Enfermagem, Faculdade Estácio de Sergipe (FASE) – Aracaju (SE)

Acadêmica de enfermagem bacharelado 10º período; Bolsista dos programas PIBIC E PIIC; Ligante da liga de cardiologia.

Rafaela Ribeiro Machado, urso de Enfermagem, Faculdade Estácio de Sergipe (FASE) – Aracaju (SE)

Enfermeira

José Marden Mendes Neto, Ciências Fisiológicas, Universidade Federal de Sergipe (UFS) - Aracaju (SE)

Graduado na Universidade Federal da Paraíba, especialista em auditoria de sistemas de saúde. Mestrado em Ciências fisiológicas pela Universidade Federal de Sergipe. Doutorando em Ciências Fisiológicas UFS. 

Míriam Geisa Virgens Menezes, Curso de Enfermagem, Universidade Federal de Sergipe (UFS) – Aracaju (SE)

Especialista em educação; Urgência e Emergência em Unidade de Terapia Intensiva; Doutoranda em Ciências Fisiológicas

Gilmara Beatriz Andrade da Silva, Ciências da Saúde, Universidade Federal de Sergipe (UFS) – Aracaju (SE)

Doutoranda eo Ciências Fisiológicas

Mestrado em Ciências da Saúde

Bacharel em Biomedicina pela Universidade Tiradentes

Licenciada em Ciências Biológicas

Sandra Lauton Santos, Bioquímica e Imunologia, Universidade Federal de Sergipe (UFS) – Aracaju (SE)

Pós-doutoramento em Montpellier/França no Institut National de la Santé et de la Recherche Médicale-Unité de Recherche de Physiopathologie Cardiovasculaire. Doutora em Ciências pelo Departamento de Bioquímica e Imunologia do Instituto de Ciências Biológicas/UFMG. Mestrado em Imunologia e Bioquímica pelo mesmo departamento. Bacharel em Bioquímica e Imunologia

References

Jacob KD, NorenHooten N, Trzeciak AR, Evans MK. Markers of oxidant stress that are clinically relevant in aging and age-related disease. Mech Ageing Dev. 2013;134(3-4):139-57. http://dx.doi.org/10.1016/j.mad.2013.02.008

Gasparovic AC, Jaganjac M, Mihaljevic B, Sunjic SB, Zarkovic N. Assays for the measurement of lipid peroxidation. Methods Mol Biol. 2013;965:283-96. http://dx.doi.org/10.1007/978-1-62703-239-119

McCord JM. The evolution of free radical and oxidative stress. Am J Med. 2000;108(8):652-9. https://dx.doi.org/10.1016/S0002-9343(00)00412-5

Sterba M, Popelová O, Vávrová A, Jirkovský E, Kovaríková P, Gerš V, et al. Oxidative stress, redox signaling, and metal chelation in anthracyclinecardiotoxicity and pharmacological cardioprotection. Antioxid Redox Signal. 2013;18(8):899-929. https://dx.doi.org/10.1089/ars.2012.4795

Nordberg J, Arner ESJ. Reactive oxygen species, antioxidant and the mammalian thioredoxin system. Free Rad Biol and Med. 2001;31(11):1287-312. https://dx.doi.org/10.1016/S0891-5849(01)00724-9

Barbosa KBF, Costa NMB, Alfenas RCG, Paula SO, Minim VPR, Bressan J. Estresse oxidativo: conceito, implicações e fatores modulatórios. Rev Nutr. 2010;23(4):629-43. http://dx.doi.org/10.1590/S1415-52732010000400013

McCord, JM.The evolution of free radicals and oxidative stress. Am J Med. 2000;108(8):652-9. http://dx.doi.org/10.1016/S0002-9343(00)00412-5

Betteridge, DJ. What is oxidative stress? Metabolism. 2000;49(2 Suppl 1):3-8. http://dx.doi.org/10.1016/S0026-0495(00)80077-3

Halliwell B. Oxidants and human disease: some new concepts. FASEB J. 1987;1(5):358-64. https://dx.doi.org/10.1096/fasebj.1.5.2824268

Barreiros ALBS, David JM, David JP. Estresse oxidativo: relação entre geração de espécies reativas e defesa do organismo. Quím Nova. 2006;29(1):113-23. http://dx.doi.org/10.1590/S0100-40422006000100021

Cook NC, Sammans S. Flavonoids- Chemistry, metabolism, cardioprotective effects, and dietary sources. J Nutr Biochem. 1996;7:66-76. https://dx.doi.org/10.1016/S0955-2863(95)00168-9

Tahir M, Rehman MU, Lateef A, Khan AQ, Khan R, Qamar W, et al. Diosmin abrogate schemically induced hepatocarcinogenesis via alleviation of oxidative stress, hyperproliferative and inflammatory markers in murine model. Toxicol Lett. 2013;220(3):205-18. http://dx.doi.org/10.1016/j.toxlet.2013.04.004

Tamamoto LC1, Schmidt SJ, Lee SY. Sensory profile of a model energy drink with varying levels of functional ingredients-caffeine, ginseng, and taurine. J Food Sci. 2010;75(6):S271-8. https://dx.doi.org/10.1111/j.1750-3841.2010.01655.x

Craig WJ. Health promoting properties of common herbs. Am J Clin Nutr. 1999;70(Suppl.3):491s-9. https://dx.doi.org/10.1111/10.1093/ajcn/70.3.491s

Silambarasan T, Raja B. Diosmin, a bioflavonoid reverses alterations in blood pressure, nitric oxide, lipid peroxides and antioxidant status in DOCA-salt induced hypertensive rats. Eur J Pharmacol. 2012;679(1-3):81-9. dx.https://dx.doi.org/10.1016/j.ejphar.2011.12.040

Ahmed S, Mundhe N, Borgohain M, Chowdhury L, Kwatra M, Bolshette N, et al. Diosmin modulates the NF-kB signal transduction pathways and downregulation of various oxidative stress markers in alloxan induced diabetic nephropathy. Inflammation. 2016;39(5):1783-97. http://dx.doi.org/10.1007/s10753-016-0413-4

Tanrikulu Y, Sahin M, Kismet K, Kilicoglu SS, Devrim E, Tanrikulu CS, et al. The protective effect of diosmin on hepatic ischemia reperfusion injury: an experimental study. Bosn J Basic Med Sci. 2013;13(4):218-24. http://dx.doi.org/10.17305/bjbms.2013.2305

Queenthy SS, John B. Diosmin exhibits anti-hyperlipidemic effects in isoproterenol induced myocardial infarcted rats. Eur J Pharmacol. 2013;718(1-3):213-8. http://dx.doi.org/10.1016/j.ejphar.2013.08.031

Rehman MU, Tahir M, Quaiyoom Khan A, Khan R, Lateef A, Hamiza OO, et al. Diosmin protects against trichloroethyleneinduced renal injury in Wistar rats: plausible role of p53, Bax and caspases. Br J Nutr. 2013;110(4):699-710. dx.https://dx.doi.org/10.1017/S0007114512005752

Senthamizhselvan, Manivannan J, Silambarasan T, Raja B. Diosmin pretreatment improves cardiac function and suppresses oxidative stress in rat heart after ischemia/reperfusion. Eur J Pharmacol. 2014;736:131-7. http://dx.doi.org/10.1016/j.ejphar.2014.04.026

Arab HH, Salama SA, Omar HA, Arafael-SA, Maghrabi IA. Diosmin protects against ethanol-induced gastric injury in rats: novel antiulcer actions. PloS One. 2015;10(3):e0122417. https://dx.doi.org/10.1371/journal.pone.0122417

Schlottfeldt FS, Fernandes SM, Martins DM, Cordeiro P, Fonseca CD, Watanabe M, et al. Prevenção da nefrotoxicidade da anfotericina B por meio do uso de fitomedicamentos. Rev Esc Enferm USP. 2015;49(esp)74-9. http://dx.doi.org/10.1590/S0080-623420150000700011

Hasan HF, Abdel-Rafei MK, Galal SM. Diosmin attenuates radiation-induced hepatic fibrosis by boosting PPAR-γ expression and hampering miR-17-5p-activated canonical Wnt-β-catenin signaling. Biochem Cell Biol. 2017;95(3):400-14. https://dx.doi.org/10.1139/bcb-2016-0142

Bondeva T, Wolf G. Reactive oxygen species in diabetic nephropathy: friend or foe? Nephrol Dial Transplant. 2014;29(11):1998-2003. https://dx.doi.org/10.1093/ndt/gfu037

Hasanoglu E, Altan N, Sindel S, Ongun CO, Bali M, Altintas E. The relation ship between erythrocyte superoxide dismutase activity and plasma levelsof some trace elements (AL, CU, ZN) of dialysis patients. Gen Pharmacol. 1994;25(1):107-10. https://dx.doi.org/10.1016/0306-3623(94)90018-3

Richard MJ,ArnaudJ,Jurkovitz C, Hachache T, Meftahi H, Laporte F, et al. Trace elements and lipid peroxidation abnormalities in patients with chronic renal failure. Nephron 1991;57:10-15. https://dx.doi.org/10.1159/000186208

Abdalla DSP. Antioxidantes. Conceitos básicos e perspectivas terapêuticas. ARS Cvrandi. 1993;26:141-64.

Halliwell B, Gutteridge JM, Cross CE. Free radicals, antioxidants, and human disease: where are we now? J Lab Clin Med. 1992;119(6):598-620.

Kim MJ, Lim Y. Protective effect of short-term genistein supplementation on the early stage in diabetes-induced renal damage. Mediators Inflamm. 2013;2013:510212. https://dx.doi.org/10.1155/2013/510212